Lab Members

Fang Zhou earned a Master’s degree in Biochemistry and Molecular Biology from the University of Alaska, Fairbanks, and joined the Germino lab at NIDDK as a biologist in 2009. She participates in various research activities including maintaining the lab infrastructure, managing the animal colony and developing complex breeding strategies, overall providing scientific and technical support to the research team.

Yu Ishimoto studied medicine at the Wakayama Medical University in Japan, from 2001 - 2007. Following his primary medical degree, he undertook residency training in internal medicine at the University of Tokyo Hospital and the Keio University Hospital. He then commenced fellowship training in nephrology at Mitsui Memorial Hospital. He studied for a Ph.D. in nephrology with focus on mitochondrial function in autosomal dominant polycystic kidney disease (ADPKD) under the supervision of Professors Masaomi Nangaku and Reiko Inagi at Graduate School of Medicine and Faculty of Medicine, The University of Toyo. Yu’s main interest is to find therapeutic options for polycystic kidney diseases.

Jiahe Qiu graduated from Tianjin Medical University in 2015 and from Tohoku University in 2018 with a Master’s Degree from its Internal Medicine and Rehabilitation Science Department. In 2022, she received her Ph. D. from the Tohoku University Graduate School of Medicine. She joined the PKD Section to learn cutting-edge methods and approaches that she intends to use to find therapeutics for Polycystic Kidney Disease.

Gyuyeong Rah joined the Polycystic Kidney Disease Section at NIDDK as a Ph.D. student in Aug 2022. She received a Bachelor’s and a Master’s degree from the Department of Biological Sciences at Sookmyung Women’s university in the Republic of Korea. Her previous research was focused on the role of primary cilia and ciliary proteins in polycystic kidney disease models. She is currently working on models of autosomal recessive polycystic kidney disease. Gyuyeong’s long-term research goal is to understand the mechanism of disease in ciliopathies.